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Androgenetic alopecia (female pattern baldness) in women is often linked to hormonal changes with the hair loss following events such as the menopause, childbirth or as a result of stopping or starting oral contraceptive pills. The hair loss is generally more uniform over the scalp than in the male counterpart, but also results from a complex chemical reaction when the enzyme 5-alpha-reductase converts the testosterone in the system into DHT or dihydrotestosterone. The hair follicles are genetically predisposed to be over sensitive to the DHT and become smaller and smaller with time, leading to the eventual hair loss.Telogen Effluvium occurs when the growing phase of the hair is interrupted prematurely causing the hair follicles to enter the telogen (resting) phase of the hair cycle earlier than normal. Two to four months later, following the normal cyclic pattern, diffuse shedding of the hair begins. When this happens there are not enough hairs left in the anagen phase, or growing phase, and the result is diffuse thinning of the hair.Pregnancy will also affect hair production.Therefore considered that minoxidil through the above cytokines to stimulate the growth of hair follicles and dermal papilla blood vessel formation and function, which plays an important role in the treatment of hair loss.In 1988, 2 percent minoxidil solution was approved topical treatment of male pattern androgenetic alopecia. 8-50 year-old male patients with mild to moderate top of the hair loss, minoxidil group number of hairs increased after 12 months of treatment, histological proof of the diameter of the hair growth by minoxidil group also increased. Applied 5% minoxidil in male pattern androgenetic alopecia, 2% Minoxidil, Seoul and placebo-controlled, by 48 months of treatment, the results of the application of 5% minoxidil group hair count than the 2% minoxidil was 45%, five times higher than in the placebo group.5% minoxidil solution can be used as male pattern hair loss.